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Naslov:Molecules involved in motility regulation in Escherichia coli cells: a review
Avtorji:ID Khan, Fazlurrahman (Avtor)
ID Tabassum, Nazia (Avtor)
ID Pham, Dung (Avtor)
ID Oloketuyi, Sandra (Avtor)
ID Kim, Young-Mog (Avtor)
Datoteke: Gradivo nima datotek, ki so prostodostopne za javnost. Gradivo je morda fizično dosegljivo v knjižnici fakultete, zalogo lahko preverite v COBISS-u. Povezava se odpre v novem oknu
Jezik:Angleški jezik
Vrsta gradiva:Delo ni kategorizirano
Tipologija:1.01 - Izvirni znanstveni članek
Organizacija:UNG - Univerza v Novi Gorici
Opis:The initial colonization of the host organism by commensal, probiotic, and pathogenic Escherichia coli strains is an important step in the development of infections and biofilms. Sensing and colonization of host cell surfaces are governed by flagellar and fimbriae/pili appendages, respectively. Biofilm formation confers great advantages on pathogenic E. coli cells such as protection against the host immune system, antimicrobial agents, and several environmental stress factors. The transition from planktonic to sessile physiological states involves several signaling cascades and factors responsible for the regulation of flagellar motility in E. coli cells. These regulatory factors have thus become important targets to control pathogenicity. Hence, attenuation of flagellar motility is considered a potential therapy against pathogenic E. coli. The present review describes signaling pathways and proteins involved in direct or indirect regulation of flagellar motility. Furthermore, application strategies for antimotility natural or synthetic compounds are discussed also.
Ključne besede:Biofilm formation, Escherichia coli, inhibition, motility, pathogenesis
Leto izida:2020
Št. strani:1-20
Številčenje:8, 36
PID:20.500.12556/RUNG-6206 Novo okno
COBISS.SI-ID:47104003 Novo okno
DOI:10.1080/08927014.2020.1826939 Novo okno
NUK URN:URN:SI:UNG:REP:FXPUJ0U7
Datum objave v RUNG:14.01.2021
Število ogledov:4895
Število prenosov:0
Metapodatki:XML DC-XML DC-RDF
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Naslov:Biofouling
Leto izida:2020

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